HitAutism.com » Archive of 'Oct, 2010'

ADHD AND SECOND HAND SMOKE

A new study suggest out of the University of California San Francisco suggest that children exposed to second hand smoke are more likely to have ADHD and to stutter. According to the CDC your risk of coronary heart disease and stroke  increases by 2 to 4 times. your risk of developing lung cancer increase by 23 and  13times respectively for males and females. Your risk of developing  COPD increases by 12 to 13 times. If you don’t care about yourself, why not quit for the kids. This study found the rates of ADHD and stuttering were nearly doubled for children ages 4 to 15 exposed to second hand smoke . Other risk factors such as being male or a familial history can not being avoided but this risk factor certainly can. Attempts to quit smoking can be extremely difficult but there is help available for those who are motivated. Perhaps this will serve as motivation for parents to get help. You may not be able to quit or even may not want to quit but if you have children especially boys please make the effort to not expose them to second hand smoke. Changing habits to include not smoking in the car with children present and stepping outside to smoke  may help prevent your child from  risk factors  associated with ADHD such as difficulty in school, higher substance abuse rate and higher incarceration rates.

Autism and Jaundice Babies: A Risk Factor

A new study in the journal of Paediatric Perinatal Epidemiology finds an association between babies that have Autism and babies born with jaundice. Babies born with jaundice have  nearly fourfold risk of developing Autism. Jaundice or hyperbilirubinemia  occurs when bilirubin ( a product of the breakdown of red blood cells) can not be cleared sufficiently by the liver. This condition is normally treated by with photo therapy. Approximately 50% of babies are born with jaundice but only about 5% require treatment. Please be cautious as this is only a risk factor and not a marker. Other risk factors include parental age, familial history, male child,breech presentation, birth before 35 weeks, low birth weight, mothers who used medicine during pregnancy. This study also found a strong association between neurological  signs, particularly hypertonicity ( too much muscle tone) and autism.  It is interesting to note that the liver is involved in detoxification a process  which many believe is a problem with  children on the autism spectrum and that jaundice involves the liver. As metal detoxification  ( mercury and aluminum) are always the topic ( vaccines and autism) in the Autism community it would be interesting to see of those children with autism who had jaudice, how many reported symptoms after the vaccine. I am of the opinion that it is a total load problem that causes the child to decompensate. Total load being the summation of stressors on the child  that cause the breakdown. Thus if 1000 children recieved the vaccine and the vaccine was the only cause then all 1000 would develop an autism spectrum disorder. This does not occur as perhaps it is only the child with the unknown infection or in this case under functioning liver that breaks the camel back and decompensates the child.  Or as in the recent legal case a child with an unknown mitochondrial issue.
Thus the take home for parents is to know your risk factors and if they start to add up be on the look out for early signs. Early detection leads to early treatment which is always best.
 
 
Paediatr Perinat Epidemiol. 2008 Nov;22(6):562-8.

Neonatal jaundice: a risk factor for infantile autism?

Maimburg RD, Vaeth M, Schendel DE, Bech BH, Olsen J, Thorsen P.

Department of Epidemiology, Institute of Public Health, University of Aarhus, Aarhus, Denmark. rmai@soci.au.dk

Abstract

In a previous study, we found that infants transferred to a neonatal ward after delivery had an almost twofold increased risk of being diagnosed with infantile autism later in childhood in spite of extensive controlling of obstetric risk factors. We therefore decided to investigate other reasons for transfer to a neonatal ward, in particular hyperbilirubinaemia and neurological abnormalities. We conducted a population-based matched case-control study of 473 children with autism and 473 matched controls born from 1990 to 1999 in Denmark. Cases were children reported with a diagnosis of infantile autism in the Danish Psychiatric Central Register. Conditional logistic regression was used to calculate odds ratios (OR) and 95% confidence intervals [CI] and likelihood ratio tests were used to test for effect modification. We found an almost fourfold risk for infantile autism in infants who had hyperbilirubinaemia after birth (OR 3.7 [95% CI 1.3, 10.5]). In stratified analysis, the association appeared limited to term infants (>or=37 weeks gestation). A strong association was also observed between abnormal neurological signs after birth and infantile autism, especially hypertonicity (OR 6.7 [95% CI 1.5, 29.7]). No associations were found between infantile autism and low Apgar scores, acidosis or hypoglycaemia. Our findings suggest that hyperbilirubinaemia and neurological abnormalities in the neonatal period are important factors to consider when studying causes of infantile autism.

PMID: 19000294 [PubMed - indexed for MEDLINE]